Publications of RA Batista

Journal Article (2)

1.
Journal Article
Batista, R.; Wang, L.; Bogaert, K.; Coelho, S.: Insights into the molecular bases of multicellular development from brown algae. Development 151 (20), dev.203004 (2024)
2.
Journal Article
Castric, V.; Batista, R.; Carré, A.; Mousavi, S.; Mazoyer, C.; Godé, C.; Gallina, S.; Ponitzki, C.; Theron, A.; Bellec, A. et al.; Marande, W.; Santoni, S.; Mariotti, R.; Rubini, A.; Legrand, S.; Billiard, S.; Vekemans, X.; Vernet, P.; Saumitou-Laprade, P.: The homomorphic self-incompatibility system in Oleaceae is controlled by a hemizygous genomic region expressing a gibberellin pathway gene. Current Biology 34 (9), pp. 1967 - 1976 (2024)

Meeting Abstract (1)

3.
Meeting Abstract
Wang, L.; Liu, P.; Lotharukpong, S.; Zheng, M.; Luthringer, R.; Batista, R.; Haas, F.; Coelho, S.: Exploring The Emergence And Evolution Of Multicellularity In The Brown Algae. In EURO EVO DEVO 2024, S08.3, p. 126. European Society for Evolutionary Developmental Biology EURO EVO DEVO 2024 (EED 2024), Helsinki, Finland, June 25, 2024 - June 28, 2024. (2024)

Poster (2)

4.
Poster
Batista, R.; Baduel, P.; Delannoy, E.; Castric, V.; Barois, M.: Transcriptional regulation of dominance at the self-incompatibility locus in Arabidopsis. Plant Epigenetics: EPIPLANT Society for Experimental Biology, Clermont-Ferrand, France (2024)
5.
Poster
Barois, M.; Batista, R.; Baduel, P.; Delannoy, E.; Castric, V.: Transcriptional regulation of dominance at the self-incompatibility locus in Arabidopsis. International Congress on Transposable Elements (ICTE 2024), Saint Malo, France (2024)

Preprint (1)

6.
Preprint
Batista, R.; Durand, E.; Mörchen, M.; Azevedo-Favory, J.; Simon, S.; Dubin, M.; Kumar, V.; Lacoste, E.; Cruaud, C.; Blassiau, C. et al.; Barois, M.; Holl, A.-C.; Ponitzki, C.; Faure, N.; Marande, W.; Vautrin, S.; Fobis-Loisy, I.; Aury, J.-M.; Legrand, A.; Krämer, U.; Lagrange, T.; Vekemans, X.; Castric, V.: Modifiers of genetic dominance at the Arabidopsis self-incompatibility locus retain proto-miRNA features and act through non-canonical gene silencing pathways. (submitted)
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